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Table 1 Prevalence of superantigen and superantigen-like open reading frames in blood and colonising isolates

From: Genotypic and phenotypic analysis of clinical isolates of Staphylococcus aureus revealed production patterns and hemolytic potentials unlinked to gene profiles and source

 

All (n = 51)a

Blood (n = 22)a

Colonising (n = 29)a

P-valueb

sea

8 (15.7)

2 (9.1)

6 (20.7)

0.26

seb

1 (1.9)

0 (0)

1 (3.4)

nd

sec

6 (11.8)

3 (13.6)

3 (10.3)

0.72

sed

1 (1.9)

0 (0)

1 (3.4)

nd

see

-

-

-

 

tst

8 (15.7)

5 (22.7)

3 (10.3)

0.23

seg

31 (60.8)

12 (54.5)

19 (65.5)

0.43

seh

3 (5.9)

1 (4.5)

2 (6.9)

0.72

sei

33 (64.7)

12 (54.5)

21 (72.4)

0.19

selj

1 (1.9)

0 (0)

1 (3.4)

nd

selk

-

-

-

 

sell

6 (11.8)

3 (13.6)

3 (10.3)

0.72

selm

33 (64.7)

12 (54.5)

21 (72.4)

0.19

seln

33 (64.7)

12 (54.5)

21 (72.4)

0.19

selo

33 (64.7)

12 (54.5)

21 (72.4)

0.19

selp

6 (11.8)

4 (18.2)

2 (6.9)

0.22

selq

-

-

-

 

selr

1 (1.9)

0 (0)

1 (3.4)

nd

sels

-

-

-

 

selt

-

-

-

 

selu c

21 (41.2)

8 (36.4)

13 (44.8)

0.54

selv

-

-

-

 

selw d

4 (7.8)

2 (9.1)

2 (6.9)

0.77

selx

39 (76.4)

17 (77.3)

22 (75.8)

0.91

  1. aNo. of PCR-amplified and sequenced genes, percentages are in parentheses
  2. bPearson’s chi-squared test (χ 2) performed for distribution of in-frame genes (n > 1)
  3. c selu and selu2 combined as selu
  4. d selw with identified ATG as start codon